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Stimulator interferon genes (STING) is another important player during antiviral responses where N-terminal domain binds/inactivates STING, thereby diminishing IRF3 activation/IFN secretion (34)
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The impaired adipogenesis increases risk of IR and T2DM by triggering ectopic fat deposition in nonadipose tissues and proinflammatory environment characterized by impaired secretion of various adipose-derived adipokines [13]
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